library.onepin.app/injectable-vitamin-c Peptide Education Library
Education only, not medical advice 21+ Every dose is an example to finalize with a licensed provider

OnePin Peptide Library · Metabolic

Metabolic Peptide Not FDA-approved · investigational

Injectable Vitamin C

IV Vitamin C · Ascorbic Acid (Injectable)

Injectable vitamin C (ascorbic acid) is a parenteral formulation of the essential water-soluble vitamin, administered intravenously (IV) or intramuscularly (IM). Vitamin C is a potent antioxidant and essential cofactor for numerous enzymatic reactions including collagen synthesis, carnitine biosynthesis, and neurotransmitter production. At physiological concentrations, it functions primarily as an antioxidant, but at the supraphysiological plasma levels achievable only through IV administration (>200mg/dL), it exhibits pro-oxidant properties that have been investigated in oncology research.,IV vitamin C can achieve plasma concentrations 30-70 times higher than the maximum achievable through oral supplementation, due to saturable intestinal absorption and renal threshold mechanisms. This pharmacokinetic difference is the primary rationale for injectable administration in clinical and research settings. High-dose IV vitamin C (15-100g) is used in integrative oncology, critical care (sepsis), and severe deficiency states (scurvy), while lower IM doses (500mg-2g) are used for general repletion and wellness.,The clinical evidence base is mixed. IV vitamin C has shown some promise in sepsis trials (CITRIS-ALI, VITAMINS) with inconsistent results, and early-phase oncology studies suggest possible quality-of-life benefits in advanced cancer patients. For scurvy and severe deficiency, injectable vitamin C is well-established. The compound is generally well-tolerated but requires G6PD screening before high-dose use due to risk of hemolytic anemia in deficient individuals.

Quick Start
Route
Intravenous (IV) or Intramuscular (IM)
Start low
1-25g IV or 500mg-2g IM
Frequency
1-3x weekly
Timing
No specific requirement

Start here. Every protocol below is ordered lowest-first. Begin at the smallest effective dose, hold about a week to assess tolerance, and step up only if needed.

Tiered Protocols · lowest first
Quick Start
Conservative start
1-25g IV or 500mg-2g IM · Intravenous (IV) or Intramuscular (IM) · 1-3x weekly
Lowest starting point. Hold about a week to assess tolerance before stepping up.
1-25g IV or 500mg-2g IM
1-3x weekly
Intermediate
Immune & Antioxidant Flush
500-1000 mg · IM · 2x Weekly
human study · Padayatty et al. 2004 (Ann Intern Med)
500-1000 mg
2x Weekly
01

How it works

As an electron donor, ascorbic acid serves as a cofactor for prolyl and lysyl hydroxylases (essential for collagen synthesis), dopamine beta-hydroxylase (norepinephrine synthesis), and various dioxygenases involved in carnitine biosynthesis and gene regulation via epigenetic demethylation.,At pharmacological IV concentrations (>200mg/dL plasma), ascorbic acid generates hydrogen peroxide extracellularly through auto-oxidation. Cancer cells, which often have low catalase activity, are selectively vulnerable to this oxidative stress, while normal cells with adequate catalase can neutralize H2O2. This is the basis for high-dose IV vitamin C research in oncology.,Ascorbic acid supports immune function by enhancing neutrophil chemotaxis, phagocytosis, and oxidative burst capacity. It also supports lymphocyte proliferation and antibody production, and protects immune cells from oxidative damage during inflammatory responses.

As an electron donor, ascorbic acid serves as a cofactor for prolyl and lysyl hydroxylases (essential for collagen synthesis), dopamine beta-hydroxylase (norepinephrine synthesis), and various dioxygenases involved in carnitine biosynthesis and gene regulation via epigenetic demethylation.,At pharmacological IV concentrations (>200mg/dL plasma), ascorbic acid generates hydrogen peroxide extracellularly through auto-oxidation. Cancer cells, which often have low catalase activity, are selectively vulnerable to this oxidative stress, while normal cells with adequate catalase can neutralize H2O2. This is the basis for high-dose IV vitamin C research in oncology.,Ascorbic acid supports immune function by enhancing neutrophil chemotaxis, phagocytosis, and oxidative burst capacity. It also supports lymphocyte proliferation and antibody production, and protects immune cells from oxidative damage during inflammatory responses.

02

What to expect

Early
Days 1–7

Days 1-7: Rapid plasma level elevation after IV/IM dosing. Deficiency symptoms (fatigue, poor healing) begin improving. Subjective energy boost reported by many.

03

Evidence

HumanStrong
AnimalPresent
In-vitroStrong
2021High-dose intravenous vitamin C, a promising multi-targeting agent in the treatment of cancer · Bottger F et al, Journal of Experimental & Clinical Cancer Research ↗review
2024High-Dose Intravenous Vitamin C Combined with Docetaxel in Men with Metastatic Castration-Resistant Prostate Cancer: A Randomized Placebo-Controlled Phase II Trial · Nielsen TK et al, Cancer Research Communications ↗peer reviewed
2014Intravenous Vitamin C and Cancer: A Systematic Review · Fritz H et al, Integrative Cancer Therapies ↗review
2018Systematic Review of Intravenous Ascorbate in Cancer Clinical Trials · Nauman G et al, Antioxidants ↗review
04

Interactions & stacking

What's safe to combine, and what belongs in a separate pin.

Safe & synergistic

BPC-157No pharmacological conflict - administer in separate syringes at different sites
EpithalonAntioxidant support may complement telomerase activation - no adverse interaction
TB-500No direct interaction - vitamin C supports collagen synthesis while TB-500 promotes tissue repair
GlutathioneVitamin C regenerates oxidized glutathione (GSSG) back to reduced form (GSH) - synergistic antioxidant recycling

Keep separate

Injectable B-ComplexAscorbic acid degrades cyanocobalamin (B12) when mixed - never combine in the same syringe
Any compound in same syringeInjectable vitamin C is acidic (low pH) and can denature peptides and degrade other compounds - always administer alone
GHK-CuVitamin C is a reducing agent that can reduce Cu2+ to Cu1+, disrupting the copper-peptide complex essential for GHK-Cu activity
Methylene BlueVitamin C can reduce methylene blue to leucomethylene blue, altering its redox cycling and potentially negating its mitochondrial electron carrier function
05

Pharmacokinetics

Illustrative plasma concentration · 24 h
Half-life
~30 min (IV), ~2 hours (IM). Renal elimination with dose-dependent kinetics.
Peak · Tmax
Immediate (IV), ~30-60 min (IM)
Elimination
~2-4 hours (IV), ~8-10 hours (IM). Faster at high doses due to renal threshold excretion.
Activity
4-8 hours (tissue antioxidant effect persists longer)

Storage. Store refrigerated at 2-8C. Protect from light. Ascorbic acid is sensitive to oxidation; discard if solution is darkened or discolored.

06

Regulatory status

FDA-approved for treatment and prevention of vitamin C deficiency (scurvy) and conditions requiring parenteral supplementation. High-dose IV use in oncology and sepsis is investigational/off-label.

Put it to work

Calculate, log & track

Open Injectable Vitamin C straight into OnePin with your vial and dose pre-filled, then let it handle the syringe math and remind you before the vial runs out.

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Go deeper

The guide & community

The complete Injectable Vitamin C walkthrough, real protocol discussion and coaching live inside the BlessUp community on Skool.

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