library.onepin.app/halotestin-fluoxymesterone Peptide Education Library
Education only, not medical advice 21+ Every dose is an example to finalize with a licensed provider
Hormonal Anabolic-Androgenic SteroidFDA-approved medicineControlled substancePrescription drug

Halotestin (Fluoxymesterone)

Fluoxymesterone

10mg – 10-40 mg/day Oral 1-2x daily
C17-alpha-alkylated OralTestosterone DerivativeNon-AromatizableHigh-Androgen

Fluoxymesterone, marketed historically as Halotestin, is an orally active anabolic-androgenic steroid derived from testosterone. It is a C17-alpha-alkylated compound with fluorine at the 9-alpha position and a hydroxyl at 11-beta, giving it high oral bioavailability and very high androgenic potency. Developed by Upjohn and introduced in 1957, it was approved for hypogonadism, delayed puberty, and later for breast cancer in women. It is a Schedule III controlled substance in the United States.

Quick Start
Route
Oral
Start low
10mg Oral
Frequency
1-2x daily
Timing
Take with food to reduce GI upset

Split daily dose AM and pre-training given the ~9 hour half-life. Maximum cycle duration 2-4 weeks due to severe hepatotoxicity. Liver enzymes MUST be checked pre and post. Have a testosterone base running - halotestin suppresses endogenous testosterone rapidly.

Start here. Every protocol below is ordered lowest-first. Begin at the smallest effective dose, hold about a week to assess tolerance, and step up only if needed.

Tiered Protocols · lowest first
Conservative start
10mg · Oral · 1-2x daily
conservative starter
10mg
1-2x daily
Expert
Pre-Contest / Peak Strength
10-20 mg · Oral · Daily
anecdotal · community
Does not aromatize to estrogen. Provides intense neurological drive and muscle density without water weight.
10-20 mg
Daily
Research
Metastatic Breast Cancer (FDA label)
10-40 mg/day · Oral · Divided 3-4x daily
FDA labeling + human clinical trials · FDA Halotestin label; Piedmont Oncology phase II 1992 (Am J Clin Oncol, PMID 1590276)
10-40 mg/day
Divided 3-4x daily
01

How it works

Fluoxymesterone binds the androgen receptor (AR) with very high affinity and drives strong androgenic signaling. Its anabolic contribution to muscle mass is relatively modest compared to other AAS, but its effect on the central nervous system, aggression, and strength expression is pronounced.

It does not aromatize to estrogen due to the 9-alpha-fluorine substitution, but it has weak progestogenic activity and may bind the progesterone receptor at high doses. It does not produce water retention, and gyno risk is low from fluoxymesterone itself.

The C17-alpha-alkyl group protects it from first-pass hepatic metabolism (high oral bioavailability) but also imparts severe hepatotoxicity - among the worst of any AAS. Cholestatic jaundice and hepatic lesions have been documented with relatively short courses. Lipid shift (severe HDL drop, severe LDL rise) is also extreme. Endogenous testosterone suppression is rapid and complete.

02

What to expect

Early
Days 1–7

First 3-5 days: noticeable mood and aggression change, mental intensity increase. Strength in the gym begins to rise quickly.

Mid
Weeks 2–4

Week 2-3: peak strength effect on competitive lifts. Mental intensity peaks. Cardiovascular and hepatic stress accumulating.

Later
Weeks 4–12

Week 3-4 (end of cycle): sustained strength but diminishing marginal return. Strongly advised to stop at 4 weeks maximum due to hepatic and lipid burden.

03

Evidence

HumanPresent
AnimalStrong
In-vitroPresent

References for this compound are being re-checked against their source records. We are not showing them until each one is confirmed to support the claim it sits under.

04

Side effects & safety

Commonly reported

  • Elevated liver enzymes (ALT/AST) - often severe, 3-6x ULN within 2-4 weeks
  • Severe lipid shift (HDL drop 30-50%, LDL rise 30-50%)
  • Marked aggression and mood changes
  • Suppression of endogenous testosterone (rapid and complete)
  • Oily skin and acne

Less common & notes

  • Hair loss acceleration (severe in genetically predisposed users), insomnia, anxiety, headaches, elevated blood pressure, nausea.
  • Women: very high virilization risk - contraindicated except for historical supervised breast cancer use.
  • Rare but serious: cholestatic jaundice (can occur after 2-3 weeks), hepatic adenoma, peliosis hepatis, hepatocellular carcinoma with prolonged use, severe cardiovascular events from lipid shifts, violent behavior episodes in susceptible users.
05

Pharmacokinetics

Illustrative plasma concentration · 38 h
Half-life
~9 hours
Peak · Tmax
1-2 hours
Elimination
2-3 days
Bioavailability
~80-90%

Oral

Duration of action
10-14 hours

Per dose - supports 1-2x daily dosing

Clearance

Hepatic (CYP3A4, CYP2C9). Renal excretion of metabolites.

Storage. Store at controlled room temperature (20-25°C / 68-77°F). Protect from moisture and light. Keep in original container with desiccant when available. Keep out of reach of children and in a locked location - Schedule III controlled substance in the United States.

06

Regulatory status

Schedule III controlled substance in the United States under the Anabolic Steroids Control Act of 1990. Historically FDA-approved (brand name Halotestin, Upjohn/Pharmacia) for hypogonadism, delayed puberty, and advanced breast cancer. The brand was discontinued though generic fluoxymesterone may still be available by prescription. Possession or distribution without a valid prescription is a federal offense in the United States.

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The guide & community

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